Delayed post-hypoxic leukoencephalopathy
نویسندگان
چکیده
منابع مشابه
Injury of ascending reticular activating system associated with delayed post-hypoxic leukoencephalopathy: a case report
BACKGROUND Delayed post-hypoxic leukoencephalopathy (DPHL) is a demyelinating syndrome characterized by neurological relapse after an initial recovery from hypoxic brain injury. We describe a patient with impaired consciousness following DPHL, concurrent with injury of the ascending reticular activating system (ARAS) shown using diffusion tensor tractography (DTT). CASE PRESENTATION A 50-year...
متن کاملDelayed Post-Hypoxic Leukoencephalopathy: Case Report with a Review of Disease Pathophysiology
Delayed post-hypoxic leukoencephalopathy is a rare clinical phenomenon usually observed in a small number of carbon monoxide poisoning survivors. A similar phenomenon is reported here in a patient who successfully recovered from a large overdose of diazepam and methadone, but then abruptly declined 3 weeks after the initial event. Magnetic resnance revealed confluent white matter hyperintensity...
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Hypoxic ischemic encephalopathy may cause early deep white matter abnormalities on MR imaging that usually progress to include gray matter and basal ganglia change. Toxic leukoencephalopathy due to heroin inhalation predominantly causes cerebellar and posterior cerebral radiologic change. Both conditions rarely present clinically and radiologically in a delayed manner with subsequent recovery. ...
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Objective: A 33-year-old man, after substances overdose developed acute respiratory failure. Consequences of this are delirium and bilateral hypoxic damage to globus pallidus in MRI. Forty days after remission, his mental status worsened quickly. Patient showed grossly disorganized thinking and behaviour, severe memory impairment. Neurological examination revealed diffuse hypertonia and brisky ...
متن کاملDelayed restricted diffusion in carbon monoxide leukoencephalopathy.
to a wide range of neurological symptoms, ranging from headaches, dizziness and altered mental status to coma. The basis for this is thought to be hypoxia. Carbon monoxide competes with oxygen for binding to hemoglobin (Hb) and once bound, shifts the oxyhemoglobin dissociation curve to the left, thus impairing off-loading of oxygen molecules, leading to hypoxia . A sizable minority of CO poison...
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ژورنال
عنوان ژورنال: Kazan medical journal
سال: 2019
ISSN: 2587-9359,0368-4814
DOI: 10.17816/kmj2019-985